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2024/6/28 10:53:43 | Browse: 90 | 下载: 88
Publication Name |
世界华人消化杂志 |
手稿编号 |
39845 |
手稿来源省、自治区或特别行政区 |
河北省 |
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Received |
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2024-02-06 07:40 |
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Peer-Review Started |
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2024-02-06 07:59 |
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To Make the First Decision |
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Return for Revision |
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2024-02-27 07:37 |
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修回稿 |
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2024-03-08 11:56 |
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Second Decision |
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2024-05-27 01:50 |
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期刊总编辑接受 |
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公司总编辑接受 |
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2024-05-28 01:10 |
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预出版 |
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2024-05-28 01:10 |
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缴纳版面费 |
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Edit the Manuscript by Language Editor |
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排版 |
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2024-06-27 07:02 |
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在线出版 |
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2024-06-28 10:53 |
ISSN |
1009-3079 (print) and 2219-2859 (online) |
开放获取 |
This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/ |
版权 |
© The Author(s) 2024. Published by Baishideng Publishing Group Inc. All rights reserved. |
Article Reprints |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/247
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Permissions |
For details, please visit: http://www.wjgnet.com/bpg/gerinfo/207
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Publisher |
Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA |
网址 |
http://www.wjgnet.com |
专业类型 |
消化内科学 |
手稿栏目类型 |
述评 |
文章标题 |
TL1A作为炎症性肠病治疗靶点的研究进展
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手稿来源 |
约稿 |
所有作者列表 |
战蓉蓉, 王冬, 张晓岚 |
ORCID |
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基金项目及其编号 |
基金项目名称 |
基金编号 |
TL1A对炎症性肠病肠道树突状细胞迁移及抗原递呈功能的影响 |
81870381 |
TL1A在炎症性肠病相关肠纤维化中的作用与机制研究 |
81370501 |
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通讯作者 |
张晓岚, 博士, 教授, 主任医师, 050035, 河北省石家庄市黄河大道80号, 河北医科大学第二医院东院区消化内科. xiaolanzh@126.com |
关键词 |
TL1A; 炎症性肠病; 肠纤维化; 治疗靶点; 专家述评 |
核心内容提要 |
肿瘤坏死因子样配体1(tumor necrosis factor-like ligand 1 aberrance, TL1A)通过结合其功能性受体-死亡受体3参与维持肠道稳态, 促进慢性炎症反应的发生和发展, 促进黏膜的炎性反应及肠壁纤维化的发生. 本文将从TL1A作为炎症性肠病的治疗靶点进行评述. |
出版日期 |
2024-06-28 10:53 |
引文著录 |
战蓉蓉, 王冬, 张晓岚. TL1A作为炎症性肠病治疗靶点的研究进展. 世界华人消化杂志 2024; 32(6): 397-404 |
URL |
https://www.wjgnet.com/1009-3079/full/v32/i6/397.htm |
DOI |
https://dx.doi.org/10.11569/wcjd.v32.i6.397 |
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